GHK-Cu vs NAD+: Two Longevity Therapies That Target Aging Differently Perfect B, Doral FL
It upregulates genes involved in collagen synthesis (COL1A1, COL3A1, COL5A1), extracellular matrix remodeling (decorin, fibromodulin), and antioxidant defense (superoxide dismutase, glutathione peroxidase) while simultaneously suppressing genes associated with inflammation (IL-6, TNF-alpha), fibrinogen production, and tissue destruction (several MMPs)
Placebo effect A measurable improvement in patients receiving an inactive treatment, caused by their belief that they are receiving real treatment
Our approach integrates peptide protocols alongside traditional care to address the root biology of the conditions we treat from enamel erosion and periodontal breakdown to TMJ myofascial pain and chronic oral inflammation